Oral Cancer/ Age, Gender, race, and Ethnicity

    The demographics of those who develop this cancer have been consistent for some time. While historically the majority of people are over the age of 40 at the time of discovery, it’s now occurring more frequently in those under this age. Exact causes for those affected at a younger age are now becoming clearer in peer reviewed research, revealing a viral etiology (cause), the human papilloma virus number 16. There are also links to young men and women who use conventional 'smokeless' chewing or spit tobacco. Promoted by the same as a safer alternative to smoking it has in actuality not proven to be any safer to those who use it when referring to oral cancer.

     Campaigns to promote the safety of smokeless are being initiated, but it is clear that while it may reduce lung cancers, it has a negative effect on the rates of oral cancers, pancreatic cancer, periodontal disease, and the chronic infections that it produces may link to heart disease. The gains against lung cancer may occur, but there will be new losses in other areas. The jury is out on other new smokeless tobacco dissolvable products, and until their use has acceptable research behind it we recommend avoiding it. It is also confirmed that in a younger age group, including those who have never used tobacco products, have a cause which is a HPV16 viral based.

     The human papilloma virus partially vision 16, has now been shown to be sexually transmitted between partners, and is conclusively implicated in the increasing incidence of young non smoking oral cancer patients. these public statistics do not conceder such social economic factors as income levels education availability of proper health care and the increased use of both tobacco and alcohol by different ethnic populations, but all of these factors likely play as role in who develops diseases. 

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